Varicose disease is the most common disease on earth.
The venous circulation in the lower limbs is all connected.
The venous circulation displays the greatest number of anatomical variations in the whole organism.
The venous circulation in the lower limbs forms part of a pump, the main function of which is to return the venous blood to the heart.
The fact that the lower limb acts as a pump means that the veins should not be visible.
If a pump is to work efficiently, it must be able to empty its aspiration pipes.
Varicose disease must be seen from a biological point of view, not a mechanistic one.
Miopragia is the biological cause of varicose disease.
Treatment must act on the walls of the veins of the entire superficial, perforating and communicating circulation.
The hemodynamic hypertension in the limb must be treated, not individual varicose veins.
There is no etiopathogenic difference between a large varix and a telangiectasia.
A large varix and a telangiectasia are under the same hemodynamic hypertension.
The visible veins and capillaries constitute the escape valves for hemodynamic hypertension.
Severe hemodynamic hypertension may manifest itself as a single large varix.
Traditional treatment methods act on the effect of the disease, not on its cause.
Phlebectomy and sclerotherapy remove the escape valve for hemodynamic hypertension.
A three-dimensional pathology can only be treated adequately by a three-dimensional method.
Three-dimensional treatment extends to the perforating and communicating circulation in the three regions of the limb.
The objective is to restore the form and function of the venous circulation.
The veins are treated.
Treatment follows the opposite pathway to that of the formation of varicose veins.
The great saphenous vein is innocent.
Many people are born without valves in the saphenous vein, without subsequently developing varicose disease.
Reducing the caliber of the vein reduces hydrostatic thrust, which causes ulcers due to stasis.
Reducing the caliber of the vein increases flow velocity and reduces lateral pressure. The organism therefore regains a considerable amount of blood for the trophism and oxygenation of the tissues and organs.
Telangiectasias do not form as a result of hindered drainage, but for hemodynamic reasons.
If the walls of the reticular veins dilate easily, varices form and telangiectasias do not appear.
If the reticular veins do not dilate, telangiectasias will form.
If we obliterate or remove varices without treating hemodynamic hypertension, telangiectasias will form (matting)
If we obliterate or remove all the varices without treating hemodynamic hypertension, we may cause nonvisible venous dilation.
The visible vessels are the “gateways” through which the perforating and communicating veins can be properly treated.
If the “gateways” are small, the amount of solution that can be injected will be less. This makes treatment more difficult and increases the number of sessions required.
The valves of the saphenous vein do not break up hydrostatic pressure.
Hydrostatic pressure does not increase; it is fixed.