Some key concepts

  • Varicose disease is the most common disease on earth.
  • The venous circulation in the lower limbs is all connected.
  • The venous circulation displays the greatest number of anatomical variations in the whole organism.
  • The venous circulation in the lower limbs forms part of a pump, the main function of which is to return the venous blood to the heart.
  • The fact that the lower limb acts as a pump means that the veins should not be visible.
  • If a pump is to work efficiently, it must be able to empty its aspiration pipes.
  • Varicose disease must be seen from a biological point of view, not a mechanistic one.
  • Miopragia is the biological cause of varicose disease.
  • Treatment must act on the walls of the veins of the entire superficial, perforating and communicating circulation.
  • The hemodynamic hypertension in the limb must be treated, not individual varicose veins.
  • There is no etiopathogenic difference between a large varix and a telangiectasia.
  • A large varix and a telangiectasia are under the same hemodynamic hypertension.
  • The visible veins and capillaries constitute the escape valves for hemodynamic hypertension.
  • Severe hemodynamic hypertension may manifest itself as a single large varix.
  • Traditional treatment methods act on the effect of the disease, not on its cause.
  • Phlebectomy and sclerotherapy remove the escape valve for hemodynamic hypertension.
  • A three-dimensional pathology can only be treated adequately by a three-dimensional method.
  • Three-dimensional treatment extends to the perforating and communicating circulation in the three regions of the limb.
  • The objective is to restore the form and function of the venous circulation.
  • The veins are treated.
  • Treatment follows the opposite pathway to that of the formation of varicose veins.
  • The great saphenous vein is innocent.
  • Many people are born without valves in the saphenous vein, without subsequently developing varicose disease.
  • Reducing the caliber of the vein reduces hydrostatic thrust, which causes ulcers due to stasis.
  • Reducing the caliber of the vein increases flow velocity and reduces lateral pressure. The organism therefore regains a considerable amount of blood for the trophism and oxygenation of the tissues and organs.
  • Telangiectasias do not form as a result of hindered drainage, but for hemodynamic reasons.
  • If the walls of the reticular veins dilate easily, varices form and telangiectasias do not appear.
  • If the reticular veins do not dilate, telangiectasias will form.
  • If we obliterate or remove varices without treating hemodynamic hypertension, telangiectasias will form (matting)
  • If we obliterate or remove all the varices without treating hemodynamic hypertension, we may cause nonvisible venous dilation.
  • The visible vessels are the “gateways” through which the perforating and communicating veins can be properly treated.
  • If the “gateways” are small, the amount of solution that can be injected will be less. This makes treatment more difficult and increases the number of sessions required.
  • The valves of the saphenous vein do not break up hydrostatic pressure.
  • Hydrostatic pressure does not increase; it is fixed.